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It was previously reported that targeted FUS-induced BBB opening facilitates CNS penetration of systemically administered anti-A antibodies to targeted brain regions in the TgCRND8 and APP23 mouse model of AD, therefore reducing plaque load 5 , therefore confirms the feasibility of this strategy in promoting behavioral and memory recovery in a transgenic animal model 6,7

The G-site is located at the N-terminus and is the specific binding site for glutathione (GSH)

However, long-term oral NAC administration is associated with higher risk of RA [141], and potential clinical application of NAC requires further study

doi: 10.1016/S0014-5793(01)02652-7, 29 ClabautM.SuetA.RacineP

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