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This refined understanding underscores the necessity of cell-type-targeted therapeutic strategies that either protect vulnerable cells or modulate the response of resistant cells in cardiovascular diseases

[DOI] [PubMed] [Google Scholar] van der Slot AJ, Zuurmond A-M, Bardoel AFJ, Wijmenga C, Pruijs HEH, Sillence DO, Brinckmann J, Abraham DJ, Black CM, Verzijl N, DeGroot J, Hanemaaijer R, TeKoppele JM, Huizinga TWJ, Bank RA (2003) Identification of PLOD2 as telopeptide lysyl hydroxylase, an important enzyme in fibrosis

Key Takeaways: GSH depletion in Type 2 Diabetes is driven by impaired synthesis, not just increased oxidative consumption