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These along with telomere damage, as a result of oxidative stress, lead to upregulation of the cell cycle inhibitor p16 INK4 and mammalian target of rapamycin (mTOR)-mediated signalling to trigger premature senescence in COPD airway epithelial cells (145149)

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In celiac patients, the protein pieces cross the intestinal lining and are mistaken for dangerous particles or microorganisms

Patients with persistent systemic inflammation have a higher mortality rate and more frequent episodes of acute exacerbation ( Several studies have identified the presence of potentially pathogenic microorganisms (PPM) in the lower respiratory tract of patients with stable COPD, which may lead to systemic inflammation and increased serum C-reactive protein (CRP), interleukin-8 (IL-8) levels and plasma fibrinogen (FIB) (94, 95)

American Journal of Epidemiology