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By increasing the expression of Cyclin D1 (CCND1), Proliferating Cell Nuclear Antigen (PCNA), and BCL-2, Schisandrol B also reduces acetaminophen-induced activation of p53 and p21, thereby promoting liver regeneration (Jiang et al., 2015a)

Exogenous IGF-1 LR3 can suppress endogenous GH secretion through this feedback, potentially reducing pituitary output during the administration period [1]

Role of oxidative stress in pathophysiology of nonalcoholic fatty liver disease

& Rssner, S