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In 1977, for instance, scientists at Philip Morris had completed a draft of a paper entitled Naturally Occurring Radon-222 Daughters in Tobacco and Smoke Condensate, which the authors wanted to submit to Science
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Remember smoking kills you
The candidate genes that play a key role in nicotine addiction include those associated with the dopaminergic neurotransmitter system (e.g., DRD2, DRD3, DRD4), cellular transport system (e.g., SLC1A2, SLC6A4), serotonergic neurotransmitter system (e.g., HTR2A), nicotinic neurotransmitter system (e.g., CHRNA4, CHRNA5, CHRNA3, CHRNA7, CHRNB4), opioidergic activity (e.g., OPRM1), and nicotine metabolism (e.g., CYP2A6) ( Figure 1 nAChRs are primary targets of nicotine, nicotine exerts direct and indirect effects on other receptor systems (e.g., opioid, serotonergic, glutamatergic) that also mediate nicotine-induced behavioral and neural changes in humans
