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When your liver metabolizes alcohol, it produces free radicals, increasing oxidative stress, impacting liver function, including the ability to produce more glutathione, and depleting overall stores in the body

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[2] The same body of work clarified that dietary cysteine, cystine, methionine, N-acetylcysteine, and related precursors are effective at supporting tissue glutathione synthesis, whereas intact glutathione taken by mouth faces the additional hurdle of surviving the digestive tract before it can contribute anything