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Clinical manifestations may involve multiple systems, including neurological, gastrointestinal, and musculoskeletal domains

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These changes allow researchers to study amylin receptor signaling and related metabolic pathways over longer timeframes than would be possible with endogenous amylin alone [1]

Exosomal thioredoxin-1 from hypoxic human umbilical cord mesenchymal stem cells inhibits ferroptosis in doxorubicin-induced cardiotoxicity via mTORC1 signaling

GPx6 is closely homologous to GPx3