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Insulin-like growth factor-1 regulates proliferation and osteoblastic differentiation of calcifying vascular cells via extracellular signal-regulated protein kinase and phosphatidylinositol 3-kinase pathways

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Their therapeutic action is predicated on a confluence of mechanisms, including increased nitric oxide (NO) bioavailability, reduced oxidative stress, and the intricate modulation of vasoactive signaling cascades

293,294 Depleting macrophages or inhibiting their migration improves mechanical sensation and nerve function in animal models

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