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Clinically, EOAD progresses rapidly and may present with atypical symptoms such as visuospatial dysfunction, apraxia, or language impairment (Marshe et al., 2019

The process can be initiated by elevated glucose or insulin, and liver X receptor A, thereby promoting hepatic lipid accumulation and regulating to lipid-producing genes such as Acetyl-CoA Carboxylase (ACC), Stearoyl-CoA Desaturase-1, and Fatty Acid Synthase (FASN) (Geidl-Flueck and Gerber 2023

NMN is a precursor that must be converted by the body into NAD, and conversion efficiency varies significantly between individuals

Autism spectrum disorder in a child with propionic acidemia