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Certain nanomaterials, including metal and metal oxide nanoparticles as well as biopolymer-based nanostructures, can disrupt microbial cell membranes, generate reactive oxygen species, or interfere with cellular metabolic processes (192)

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Mechanistically, macrophage-specific inhibition of GLUL leads to accumulation of succinate and HIF-1 via glutamine-dependent -aminobutyric acid (GABA) shunting (thereby inhibiting vessel sprouting and metastasis) and via stimulation of T effector cells