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To harness this mechanism of gene upregulation, ASOs were designed to specifically target the uORF of the Lrpprc gene in mouse models and found that the treatment was successful in increasing the LRPPRC protein expression, an experiment that provided evidence to the efficacy of this mechanism [84]

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Endotoxin Control: Critical for mitochondrial assays where bacterial byproducts could skew inflammatory markers or oxygen consumption rates (OCR)