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Consistent with this paradigm, recent single-cell analyses of the corneal immune microenvironment have demonstrated that immune activation during physiological wound healing is characterized by dynamic and reversible shifts in immune cell phenotypes, whereas chronic or exaggerated responses, such as those observed in diabetic corneas, lead to maladaptive inflammation and delayed repair (Lin et al., 2025)

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However, this speculation needs to be directly verified in uterine fibroid cell models

Liu J, Xia X, Huang P