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The RNA polymerase-related factor 1 complex (Paf1C) has gradually become a key transcriptional regulatory factor in eukaryotes (Francette et al., 2021)

In order to prevent mitochondrial malfunction and sustain metabolic function/energy generation (ATP), NAD+ supplementation may counterbalance the age-related degradation of NAD+ and its precursor nicotinamide mononucleotide by NADases, particularly CD38.7 NAD+ replenishment, however, appears to support a number of other metabolic pathways via NAD+-dependent enzymes in research involving human and animal models (as well as samples and cell lines) (1,2,3)

2021;6(24):e292032

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