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A rationale for glutathione (GSH) therapy

The last one is an organo-sulfur compound first identified in broccoli [290] able to modify C151 in KEAP1, therefore inducing the antioxidant NRF2-target enzyme NQO1 [158]

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Thus, genetic engineering to enhance GPX4 expression in NK cells may prolong their survival and activity in the harsh TME, opening new avenues for NK cell-based immunotherapy (76)

[DOI] [PubMed] [Google Scholar] 228.Arriza J.L., Weinberger C., Cerelli G., Glaser T.M., Handelin B.L., Housman D.E., Evans R.M