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PRMT5 is highly expressed in breast cancer and chronic myeloid leukemia (CML) stem cells, and the knockdown of PRMT5 or the use of PRMT5 inhibitors can significantly impair the self-renewal capacity of CSCs

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We cross-referenced coimmunoprecipitation and proximity labeling data sets, which accounted for three different NHR-49 strains including the overexpressed all-tissue NHR-49::GFP under fed and fasted conditions 42 and the endogenous N- and C-terminal TurboID tagged NHR-49 48

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Patient education: Injection technique for GLP-1 RAs