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Senescence as a Modulator of Iron Metabolism and Fibrosis The hypothesized crosstalk is likely bidirectional

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In murine models, gut dysbiosis impaired the differentiation of IgA B cells into plasma cells, reducing milk sIgA levels and weakening the mammary mucosal immune barrier, thereby exacerbating S

Additionally, TiO@Ru@siRNA downregulated key immunosuppressive factors, upregulated immune cytokines, and activated CD4 and CD8 T lymphocytes, effectively remodeling the immune microenvironment and inhibiting oral cancer growth (Zhou et al