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in particular, whether the gut lung axis operates independently or is mediated in part through hepatic processing of gut-derived signals warrants further investigation

In order to confirm that the lack of induction of Ppargc1a gene expression upon combined pharmacologic PPAR and PPAR activation of is not accounted for by off-target effects of rosiglitazone and WY-14643, we infected AC16 cells with recombinant adenoviruses expressing human PPAR (Ad-PPAR) and PPAR (Ad-PPAR)

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however, the underlying pathophysiological mechanisms that mediate this relationship remain to be fully elucidated

There are a number of genetic, metabolic, and environmental factors involved in the formation and evolution of the atherosclerotic plaque (Markin, Sobenin, Grechko, Zhang, & Orekhov, 2020)