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TXNRD1 has emerged as a selective vulnerability in KRAS -wild-type and KRAS inhibitor-resistant tumors, where its blockade induces ferroptosis through combined GSH depletion and iron overload 117

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Next, an acidic amino acid residue donates a proton back to C-3, but from the opposite face, resulting in inversion at C-3 and formation of xylulose 5-phosphate

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Furthermore, the extent of modification of the putative regulatory thiol by persulfidation needs to be quantified, as low stoichiometries of modification can only be regulatory if they activate a gain-of-function of the modified protein