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Collectively, we conclude that -KG functions as an epigenetic cofactor to maintain TET-dependent DNA demethylation to facilitate YBX1 transcription, a prerequisite for AMPK translation

5FU adaptation alters mitochondrial activity Given the changes in mitochondrial abundance, without evident mitochondrial dysfunction (no cristae clearing, no change in apoptotic priming, no release of cytochrome c or mtDNA), we reasoned that the increase might be associated with increased functionality

Nassini et al., 2014)

Through the p62Keap1NRF2 pathway, NRF2 upregulates the expression of genes involved in iron and ROS metabolism, such as NQO1 , HO1 , and FTH1 , thereby suppressing ferroptosis (99)