In D-galactose-induced aging mice, oral administration of the product significantly increased NAD+ levels in liver and adipose tissue, activated mitochondrial energy metabolism pathways, improved insulin sensitivity, lowered blood glucose and lipid levels, reduced visceral fat accumulation, and increased energy expenditure, alleviating fatigue and abnormal body weight associated with aging
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Its effects stay focused on adipose tissue: AOD9604 stimulates lipolysis primarily through beta3 adrenergic receptor pathways in adipose tissue, driving the breakdown of stored triglycerides into free fatty acids
