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demonstrated that secondary metabolites produced during P

As previously described, the classical proinflammatory categories (i.e., TLR, MAPK, LPS, and IFN signaling) were enriched in the downregulated clusters (35)

Research assessing muscle and tendon healing reported increases in angiogenic markers such as VEGF, CD34, and factor VIII in injured tissue following peptide exposure [6]

Theoretically, this could result in increased fractional oxidation and an increased propensity to toxicity (see Fig

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