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Extreme restriction is unnecessary and often counterproductive, causing metabolic slowdown that persists beyond the protocol
Its mechanism of action involves multiple complementary pathways that collectively regulate glucose homeostasis, gastric function, and energy balance [4]
The break is important: it prevents receptor accommodation and allows the body to consolidate the tissue remodeling that the protocol initiated
The study posits that DSIP's actions on substance P, beta-endorphin, and corticosterone levels are part of a broader spectrum of biochemical changes, hinting that DSIP might initiate a series of molecular reactions contributing to its stress-modulatory actions
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