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Regulation is achieved also by post-translational modifications (PTMs) as phosphorylation or myristoylation and by allosteric effectors like NADPH [20]

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First identified in human plasma in the 1970s, GHK-Cu has since become one of the most studied peptides for anti-aging, wound healing, and cellular repair

Rather than acting as a uniform pathogenic signal, redox imbalance manifests with distinct temporal dynamics and biological consequences depending on disease context

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