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Description

Flow cytometric monitoring of constitutive MAPK phosphorylation and of pharmacodynamic MEK/MAPK inhibition in primary AML samples

Laboratory studies demonstrate its activity across vascular endothelial growth factor pathways and nitric oxide-dependent mechanisms[1][2]

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Differential contribution of hepatitis C virus NS5A and core proteins to the induction of oxidative and nitrosative stress in human hepatocyte-derived cells