By interacting with lectin-type Ox-LDL receptor-1, Ox-LDL is capable of suppressing the constitutive eNOS expression [107], elevating the expression of adhesion molecules and chemokines in ECs, and inducing macrophage proliferation, collagen production, vascular smooth muscle cell migration, and platelet activation [108]
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Ghani A, Barril C, Bedgood DR, et al
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