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This sensitization occurred as a consequence of parthenolide suppression of the activation of the nuclear transcription factor-kappaB (NF-B) and the sustained activation of the transcription factor regulator c-Jun N-terminal kinase (JNK)

Unlike single-pathway compounds, AHK-Cu enables researchers to investigate how mineral-peptide complexes influence multiple biological systems simultaneously, including cellular repair, follicular activity, and gene regulation

[6] Crucially for the systemic story, they then moved into in vivo rodent models