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To determine the point at which mitochondrial Fe-S protein function is compromised by cystine starvation, we assessed respiratory complex activity in cells subject to cystine deprivation for 096 h that were concomitantly treated with ferrostatin-1 to prevent the induction of ferroptosis

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Nitrosoglutathione (GSNO) and protein nitrosothiols (PSNO) are also formed in cells and play a role in NO signaling independent of the cyclic GMP pathway

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