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This synergistic interaction enhances resilience against oxidative stress and supports continuous redox cycling in experimental models of aging and cellular defense
Together, these effects disrupt the invasive, adhesive, and vascular processes required for lesion establishment and progression ( SNAIL and TWIST and apoptosis-resistance genes such as Bcl-xL , thereby improving tissue architecture and restoring apoptotic responsiveness in EMs models (Zhang et al., 2023)
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2.3 Molecular pathways regulating CL function 2.3.1 MAPK/ERK signaling The mitogen-activated protein kinase/extracellular signal-regulated kinase (MAPK/ERK) mediates a luteotrophic and luteolytic signaling in the corpus luteum (CL) ( Figure 4 )
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