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For instance, low GPx has been associated with increased oxidative damage in the brain, potentially contributing to cognitive decline

These effects are associated with increased expression of the cyclin-dependent kinase inhibitors (CDKNs) p21 and p27, which are negative cell cycle regulators (Huang et al., 2011)

Through surface modifications, we revealed that the amino, carbonyl, carboxyl, and hydroxyl on the surface of CDs affect its catalytic activity

Asperosaponin vi inhibition of dnmt alleviates gpx4 suppression-mediated osteoblast ferroptosis and diabetic osteoporosis

blocking Wnt erases the benefit, arguing that endothelial restructuring is causal, not merely correlative [183]