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The analyses were conducted using R (version 4.0.0)

In our performance medicine program, GHK-Cu is administered as a subcutaneous injection

The more reduced mitochondrial [NAD + ]/[NADH] during fasting and DR from increased neural fatty acid oxidation (low capacity in neurons) and ketone body oxidation may slow the mitochondrial malate dehydrogenase 2 (MDH2) enzyme of the MAS, inhibiting flux through the MAS leading to the more reduced cytoplasmic [NAD + ]/[NADH] observed in the brain of animals on the DR diet

GHK-Cu and semaglutide have no known drug interaction