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Apoptosis as anticancer mechanism: function and dysfunction of its modulators and targeted therapeutic strategies

-Gal A deficiency leads to the accumulation of globotriaosylceramide (Gb3) and related neutral glycosphingolipids within lysosomes, which results in impairment of cellular morphology and function 1,2

The glutathione cycle can complement the glutamate-glutamine shuttle and influence excitatory neurotransmission under conditions of glutamine restriction The glutamate-glutamine shuttle (Fig

ERK and Akt signaling pathways The three pathways activated by TrkB each serve distinct functions