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Abstract Background: Chronic liver disease is characterized by progressive hepatic glutathione depletion and oxidative stress, yet antioxidant therapies have historically shown disappointing clinical efficacy in unselected populations

Cells can stave off ferroptotic fate through the use of antioxidant pathways that largely utilize GSH, glutathione peroxidase 4 (GPX4), and perioredoxin-thioredoxin, which all ultimately rely on NADPH for regeneration

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doi: 10.1016/j.cell.2013.12.010 12 YangWSStockwellBR

Dixon, Ionization potential, electron affinity, electronegativity, hardness, and electron excitation energy: Molecular properties from density functional theory orbital energies Available from:, J