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To further verify that the decreased resistance in the hr-52 mutant is caused by the interference with AT4G23100 gene, we obtained the previously reported mutant pad2-1 with the Ser298Asn substitution (S298N) within AT4G23100 21 to assess its responses to B
In way to optimize the conduct of experiments and increase their acceptability in compliance with regulations in animal experimentation focusing on one of the 3Rs, the refinement 36,37 , we used APAP as a new therapeutic refinement tool to relieve the mice in an infectious model of toxoplasmosis
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Metabolites 11 (2), 109
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