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It has become clear that vitamin D deficiency needs to be corrected at multiple levels: (i) vitamin D supplements and/or vitamin D-rich foods need to be consumed to provide adequate vitamin D, (ii) the body needs to be able to upregulate vitamin D-metabolizing genes to convert vitamin D into 25(OH)VD and then to 1,25(OH) 2 VD, and (iii) the levels of inflammatory biomarkers need to be reduced

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To explore the molecular mechanisms underlying the protective effects of sulforaphane in the rotenone mouse model of PD, we next analysed Nrf2 pathway in both cerebral cortex and striatum

The rationale involves addressing multiple aspects of aging simultaneously through cellular protection via telomerase and metabolic support via growth hormone optimization

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