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mTORC1, which is well known for its function at the center of cancer metabolic reprogramming, promotes mitochondrial glutaminolysis via the migration of SIRT4-mediated inhibition of GLUD1 189

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doi: 10.1515/hmbci-2021-0065 173 RebarRW

Acknowledgements This work was supported by the National Natural Science Foundation of China (82360716, 82070422, and 82200330), China Postdoctoral Science Foundation (2023T160526 and 2022M722571), Research Plan Project of Shaanxi Institute of Basic Science (22JHQ053), High-end Foreign Expert Introduction Program of National Science and Technology (G2022040014L), Qinchuangyuan Traditional Chinese Medicine Innovation Research and Development Transformation Project (2022-QCYZH-036)