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*Correspondence: Lei Xu, [email protected] Liu, [email protected] These authors have contributed equally to this work This article was submitted to Cellular Neuropathology, a section of the journal Frontiers in Cellular Neuroscience Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers
Suudhof, T
These effects set the stage for a diverse array of diseases that may manifest many years later, often in response to secondary environmental stressors
Data availability The data that support the findings of this study are available upon request from the corresponding author
Similar maturational defects were observed in EcGGT when the corresponding residue Thr407 (Thr415 in BlGGT) was mutated to aspartate (T407D), lysine (T407K), and serine (T407S) (Lo et al., 2007)
