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doi: 10.3390/gidisord7040072 440 MiraghajaniMDehsoukhtehSSRafieNHamedaniSGSabihiSGhiasvandR

By integrating primary hepatocytes with non-parenchymal liver cells under continuous perfusion, the human liver-on-chip maintained long-term metabolic competence and reproduced clinically relevant mechanisms of drug-induced liver injury (DILI) at exposure levels comparable to patient plasma concentrations

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