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Indeed, Sp was able to restore cytosolic and mitochondrial redox balance in neuronal cell line SHSY-5Y subjected to 1-methyl-4-phenylpyridinium (MPP + ) treatment which is known to cause mitochondria dysfunction and oxidative stress through complex I inhibition 1 (Fig

Two potential routes for FUS-mediated plaque reduction been previously reported: (1) FUS-induced BBB opening delivered the endogenous IgG and IgM from the periphery into the brain and contributes to plaque clearance 8

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Wergeland S, Torkildsen O, Myhr KM, Mork SJ, Bo L