Neurologically, ALD/AMN can manifest with highly variable phenotypes, ranging from very aggressive and rapidly fatal inflammatory CNS demyelination in young boys to a more slowly progressive adult form either with cerebral white matter inflammatory lesions, or a more indolent progressive myelopathy and peripheral neuropathy (Moser, 1997)
Genetic and/or Nutritional Factor-Mediated Hyperhomocysteinemia and Incidence of Cardiovascular Disease In keeping with the occurrence of three major mechanisms leading to moderate hyperhomocysteinemia, Graham et al (1997) distinguish three main types of hyperhomocysteinemia using the methionine-loading test
doi:10.7326/0003-4819-153-4-201008170-00263 (2) Montgomery SR, Petrigliano FA, Gamradt SC
The fictitious group receiving 5% protein actually has a lower lesion response
[DOI] [PubMed] [Google Scholar] 81.Close RN, Schoeller DA, Watras AC, Nora EH