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Inhibition of glutathione markedly enhanced H 2 O 2 - or ARS-induced cytotoxicity in HeLa and A549 cell lines but modestly enhanced the cytotoxicity of H 2 O 2 and even did not affect the cytotoxicity of ARS in HepG2 cells

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Conclusion: Our results indicate a protective effect of IGF-1 against OA pathogenesis by inhibition of NF-B signaling via regulation of the MAPK and PI3K/Akt signaling pathways and prevention of apoptosis by suppression of ROS production

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