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5.3 Fungal and parasitic infections Emerging evidence suggests that ferroptosis represents a therapeutically exploitable pathway in both fungal and parasitic infections, with distinct intervention strategies demonstrating efficacy across different pathogen models

In in vitro systems and animal studies, modulation of these interactions has been employed to investigate cell adhesion, migration, angiogenesis-related signaling, and tissue remodeling mechanisms under experimental conditions

Activation of G protein-coupled receptor 30 protects neurons by regulating autophagy in astrocytes

ROS can induce damage to both nuclear and mitochondrial DNA through multiple mechanisms and also damage the integrity of mitochondrial permeability transition pore (mPTP), thereby aggravating the dysregulation of mitochondrial dynamics and the control of apoptosis (5052)