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1-MX is an adenosine receptor antagonist and as a major metabolite of caffeine and paraxanthine, 1-MX might contribute to their activity [19, 20]

This represents meaningful human evidence for wound healing applications, supporting the tissue repair mechanisms observed in animal models

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SSTR-mediated anti-secretory effects are induced via two main pathways: (1) inhibition of adenylyl cyclase (AC) resulting in reduced levels of cyclic AMP (cAMP) and (2) activation of K + -channels and inhibition of voltage-dependent Ca 2+ -channels resulting in reduced intracellular Ca 2+ levels (Fig