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The disorder is caused by a defective or abnormally altered gene that causes one of the proteins responsible for the initial arrest of bleeding (called von Willebrand factor) to be low or not function properly
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A different study also developed a microfluidic device to mimic human microvascular networks using PDMS to culture HUVECs and analyzed the formation of an endothelial surface layer under a physiologically relevant level of fluid shear stress [268]
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