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Substitution of two amino acids (S342G and 1,384 M) in the C terminus of the APOL1 gene causes G1 risk variants (214, 215)

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As expected, whole-cell patch-clamp electrophysiological recordings showed that PVN Glu neuronal activity was significantly higher in the AAV-DIO-shMOR-EGFP group than that in AAV-DIO-EGFP-expressing controls (Fig