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it supports cellular defense mechanisms against oxidative and metabolic stress and enhances DNA repair (Palacios et al., 2010

ACSL4 is required to inhibit GPX4-induced ferroptosis, while ALOX12 is dispensable, whereas not required in p53-mediated ferroptosis, and therefore p53 promotes ferroptosis through a GPX4-independent pathway

Ann N Y Acad Sci 786:111

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