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This sensitization to TGF-beta signaling augments expression of matrix genes including collagens, fibronectin, and proteoglycans, while appropriate regulation prevents excessive TGF-beta activity associated with pathological fibrosis

There is some evidence that such mutual exclusivity is present also in erythrocytes, complementing the situation in nucleated cells [27, 28]

Certain medicationsincluding proton pump inhibitors, metformin, and some anticonvulsantsmay affect the absorption or metabolism of specific nutrients

A genetic investigation of the essential role of glutathione: mutations in the proline biosynthesis pathway are the only suppressors of glutathione auxotrophy in yeast

10.1016/j.freeradbiomed.2020.02.027 68 Usategui-MartnR.Puertas-NeyraK.Galindo-CabelloN.Hernndez-RodrguezL