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In a mouse model of stroke, intracerebroventricular sodium selenate treatment elevates GPX4 levels by activating transcription factors activating enhancer binding protein 2 and specificity protein 1, while also providing protection against excitotoxicity and endoplasmic reticulum stress-induced cell death independent of GPX4 (102)

Melatonin reduces excitotoxic blood-brain barrier breakdown in neonatal rats

Many reported that treatment under the guidance of a clinician was preferable to self-medication, but felt they had no other choice to regain quality of life

Vitamin B-12, apolipoprotein E genotype, and cognitive performance in community-living older adults: Evidence of a gene-micronutrient interaction

10.1016/j.fertnstert.2009.12.041 114