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We feel there may be a relationship between HCCAA and other cerebral amyloid angiopathies like Alzheimers disease that is analogous to that of familial combined hypercholesterolemia (FCH) and general hypercholesterolemia

Additionally, we identify the FHA domain of SNIP1 as a reader of P-SF3B1, guiding the recruitment and assembly of the RES complex during spliceosome activation, and show that mutations in the FHA domain, including the neurodevelopmental disorder-linked E366G variant, impair this interaction, resulting in defects in splicing and cellular proliferation

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: "Is Cognitive Impairment in Cirrhotic Patients Due to Increased Peroxynitrite and Oxidative Stress?" in: Antioxidants & redox signaling , (2015) (PubMed)