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The accumulation of high levels of reactive oxygen species, neurotoxic substances, and inflammatory cytokines forms the pathological mechanism underlying CNSD, which results in dysfunction of the nervous system and the development of therapeutic targets for neurological diseases (Patel, 2016)

The sensitivity of PR3-ANCA was comparable to that of atypical perinuclear ANCA, but its specificity was significantly higher, suggesting that PR3-ANCA could serve as a potential biomarker for extensive UC [16, 33, 34]

Ayrca, serbest radikallerin neden olduu cilt hasarn nleyerek, sivilce ve dier cilt rahatszlklarnn iyileme srecini hzlandrabilir

Therefore, we hypothesize that Ruminococcus2 may produce a specific metabolite that influences the advancement of NAFLD through the gut-liver axis