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Published in vivo studies have indicated that elevated Ggt1 activity along with cellular glutathione depletion are associated with the increased oxidative stress induced by estrogen deficiency under ovariectomy or menopause conditions (Serviddio et al., 2002

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These findings have spurred interest in IGF-1 LR3 as a tool for exploring the molecular mechanisms of cellular aging and for developing strategies to promote better-supported cellular aging